Bipolar Disorder – Is it Demons????
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It does not follow absolutely that "the casting out" (itself) cured them -- Jesus could have simply done that ALSO at the same time... (It is possible.)
"Might have... Might not have..."
I imagine that (because I am not going to look them all up - just going "off of memory" here) - in most, if not all, places in scripture where an account of such is given -- the two things are probably indicated as happening at the same time without saying that the one actually caused the other...
Now, if scripture actually says that the one actually caused the other -- that is a different case...
.
"Might have... Might not have..."
I imagine that (because I am not going to look them all up - just going "off of memory" here) - in most, if not all, places in scripture where an account of such is given -- the two things are probably indicated as happening at the same time without saying that the one actually caused the other...
Now, if scripture actually says that the one actually caused the other -- that is a different case...
.
It does not follow absolutely that "the casting out" (itself) cured them -- Jesus could have simply done that ALSO at the same time... (It is possible.)
"Might have... Might not have..."
I imagine that (because I am not going to look them all up - just going "off of memory" here) - in most, if not all, places in scripture where an account of such is given -- the two things are probably indicated as happening at the same time without saying that the one actually caused the other...
Now, if scripture actually says that the one actually caused the other -- that is a different case...
.
"Might have... Might not have..."
I imagine that (because I am not going to look them all up - just going "off of memory" here) - in most, if not all, places in scripture where an account of such is given -- the two things are probably indicated as happening at the same time without saying that the one actually caused the other...
Now, if scripture actually says that the one actually caused the other -- that is a different case...
.
37 Do not believe me unless I do what my Father does. 38 But if I do it, even though you do not believe me, believe the miracles, that you may know and understand that the Father is in me, and I in the Father." John 10:37-38
U
See, Moment, here's the problem with your 'proof'. You're not asking all the right questions. Your responses are skewed, and i'm not surprised by them because the way you're asking actually sets up to support your previously held belief. You say 95% get bipolar before being saved, and that is, by some odd logic, evidence of possession.
Here's the catch to your question, the average age of onset of bipolar is late teens. That means saved or not bipolar starts that early. Many people who get saved, especially from non-Christian homes, normally don't get saved until later. So the way the facts of things play out, its actually to be expected that many bipolar cases start before salvation. So your results are skewed because you're making a black and white answer out of your question and ignoring other factors.
So you are effectively judging over 5 million US citizens (5.7 million bipolar in the US) as demon possessed and labeling them as having behavior that would lead to possession. Thats about 120 million people of our population you are accusing of involving themselves in activities that would lead them to become possessed. And if i understand right, you believe other things may also be symptoms of demon possession, so how many more millions does that add that you accuse of involving themselves in practices leading to demon possession? Are you sure you want to label over 120 million people of being possessed and performing sinful actions that lead to it? Thats quite a hefty judgement and accusation.
I'm sure you'll say you aren't judging, but you're saying these people are possessed, how did they get possessed? They had to have done something to make it happen, you can't be possessed for no reason.
Also, if you're like most Christians (which i could be wrong here, i admit i'm assuming) you don't believe Christians can be demon possessed. So assuming you believe that, then that means any Christian diagnosed with bipolar, or other things you label, are not saved. So now we're getting into judging peoples salvation. Are you sure you really want to keep going down this road under these conditions? To put yourself in such a place of judgment? Something to consider.
Here's the catch to your question, the average age of onset of bipolar is late teens. That means saved or not bipolar starts that early. Many people who get saved, especially from non-Christian homes, normally don't get saved until later. So the way the facts of things play out, its actually to be expected that many bipolar cases start before salvation. So your results are skewed because you're making a black and white answer out of your question and ignoring other factors.
So you are effectively judging over 5 million US citizens (5.7 million bipolar in the US) as demon possessed and labeling them as having behavior that would lead to possession. Thats about 120 million people of our population you are accusing of involving themselves in activities that would lead them to become possessed. And if i understand right, you believe other things may also be symptoms of demon possession, so how many more millions does that add that you accuse of involving themselves in practices leading to demon possession? Are you sure you want to label over 120 million people of being possessed and performing sinful actions that lead to it? Thats quite a hefty judgement and accusation.
I'm sure you'll say you aren't judging, but you're saying these people are possessed, how did they get possessed? They had to have done something to make it happen, you can't be possessed for no reason.
Also, if you're like most Christians (which i could be wrong here, i admit i'm assuming) you don't believe Christians can be demon possessed. So assuming you believe that, then that means any Christian diagnosed with bipolar, or other things you label, are not saved. So now we're getting into judging peoples salvation. Are you sure you really want to keep going down this road under these conditions? To put yourself in such a place of judgment? Something to consider.
Bipolar Disorders have nothing to do with demons. It has to do with medical science. There is absolutely no proof that Bipolar Disorder or any other mental disorder is linked with demonic possession.
From Bipolar disorder - Wikipedia, the free encyclopedia
Also from Bipolar Disorder Causes: Genetics, Brain Chemicals, Environment, and More
There is nothing that even mentions demonic possession for this or any mental disorder. People usually say that their mental disorder or handicap is caused by demons because of their hatred and bias against the disabled and those with a mental condition and they do not even bother to research it.
Some people claim that this is not written by someone who is saved but on the other hand, they have no right to judge anybody and therefore just because a person is saved does not mean they are incapable of error and being wrong.
It is very sad that there are those who rather judge others than to get to know them.
From Bipolar disorder - Wikipedia, the free encyclopedia
Causes
The causes of bipolar disorder likely vary between individuals. Twin studies have been limited by relatively small sample sizes but have indicated a substantial genetic contribution, as well as environmental influence. For bipolar I, the (probandwise) concordance rates in modern studies have been consistently put at around 40% in monozygotic twins (same genes), compared to 0 to 10% in dizygotic twins.[24] A combination of bipolar I, II and cyclothymia produced concordance rates of 42% vs 11%, with a relatively lower ratio for bipolar II that likely reflects heterogeneity. The overall heritability of the bipolar spectrum has been put at 0.71.[25] There is overlap with unipolar depression and if this is also counted in the co-twin the concordance with bipolar disorder rises to 67% in monozigotic twins and 19% in dizigotic.[26] The relatively low concordance between dizygotic twins brought up together suggests that shared family environmental effects are limited, although the ability to detect them has been limited by small sample sizes.[25]
Genetic
Genetic studies have suggested many chromosomal regions and candidate genes appearing to relate to bipolar disorder's development, but the results are not consistent and often not replicated.[27]
Although the first genetic linkage finding for mania was in 1969,[28] the linkage studies have been inconsistent.[29] Meta-analyses of linkage studies detected either no significant genome-wide findings or, using a different methodology, only two genome-wide significant peaks, on chromosome 6q and on 8q21.[citation needed] Neither have genome-wide association studies brought a consistent focus — each has identified new loci.[29]
Findings point strongly to heterogeneity, with different genes being implicated in different families.[30] A review seeking to identify the more consistent findings suggested several genes related to serotonin (SLC6A4 and TPH2), dopamine (DRD4 and SLC6A3), glutamate (DAOA and DTNBP1), and cell growth and/or maintenance pathways (NRG1, DISC1 and BDNF), although noting a high risk of false positives in the published literature. It was also suggested that individual genes are likely to have only a small effect and to be involved in some aspect related to the disorder (and a broad range of "normal" human behavior) rather than the disorder per se.[31]
Advanced paternal age has been linked to a somewhat increased chance of bipolar disorder in offspring, consistent with a hypothesis of increased new genetic mutations.[32]
Physiological
The causes of bipolar disorder have yet to be fully uncovered, but there is strong evidence that brain structure anomalies play a role.
Abnormalities in the structure and/or function of certain brain circuits could underlie bipolar. Meta-analyses of structural MRI studies in bipolar disorder report an increase in the volume of the lateral ventricles, globus pallidus and increase in the rates of deep white matter hyperintensities.[33][34][35] Functional MRI findings suggest that abnormal modulation between ventral prefrontal and limbic regions, especially the amygdala, likely contribute to poor emotional regulation and mood symptoms.[36]
According to the "kindling" hypothesis, when people who are genetically predisposed toward bipolar disorder experience stressful events, the stress threshold at which mood changes occur becomes progressively lower, until the episodes eventually start (and recur) spontaneously. There is evidence of hypothalamic-pituitary-adrenal axis (HPA axis) abnormalities in bipolar disorder due to stress.[37][38][39][40]
Other brain components which have been proposed to play a role are the mitochondria,[41] and a sodium ATPase pump,[42] causing cyclical periods of poor neuron firing (depression) and hypersensitive neuron firing (mania). This may only apply for type one, but type two apparently results from a large confluence of factors.[citation needed] Circadian rhythms and melatonin activity also seem to be altered.[43]
Environmental
Evidence suggests that environmental factors play a significant role in the development and course of bipolar disorder, and that individual psychosocial variables may interact with genetic dispositions.[31] There is fairly consistent evidence from prospective studies that recent life events and interpersonal relationships contribute to the likelihood of onsets and recurrences of bipolar mood episodes, as they do for onsets and recurrences of unipolar depression.[44] There have been repeated findings that between a third and a half of adults diagnosed with bipolar disorder report traumatic/abusive experiences in childhood, which is associated on average with earlier onset, a worse course, and more co-occurring disorders such as PTSD.[45] The total number of reported stressful events in childhood is higher in those with an adult diagnosis of bipolar spectrum disorder compared to those without, particularly events stemming from a harsh environment rather than from the child's own behavior.[46]
The causes of bipolar disorder likely vary between individuals. Twin studies have been limited by relatively small sample sizes but have indicated a substantial genetic contribution, as well as environmental influence. For bipolar I, the (probandwise) concordance rates in modern studies have been consistently put at around 40% in monozygotic twins (same genes), compared to 0 to 10% in dizygotic twins.[24] A combination of bipolar I, II and cyclothymia produced concordance rates of 42% vs 11%, with a relatively lower ratio for bipolar II that likely reflects heterogeneity. The overall heritability of the bipolar spectrum has been put at 0.71.[25] There is overlap with unipolar depression and if this is also counted in the co-twin the concordance with bipolar disorder rises to 67% in monozigotic twins and 19% in dizigotic.[26] The relatively low concordance between dizygotic twins brought up together suggests that shared family environmental effects are limited, although the ability to detect them has been limited by small sample sizes.[25]
Genetic
Genetic studies have suggested many chromosomal regions and candidate genes appearing to relate to bipolar disorder's development, but the results are not consistent and often not replicated.[27]
Although the first genetic linkage finding for mania was in 1969,[28] the linkage studies have been inconsistent.[29] Meta-analyses of linkage studies detected either no significant genome-wide findings or, using a different methodology, only two genome-wide significant peaks, on chromosome 6q and on 8q21.[citation needed] Neither have genome-wide association studies brought a consistent focus — each has identified new loci.[29]
Findings point strongly to heterogeneity, with different genes being implicated in different families.[30] A review seeking to identify the more consistent findings suggested several genes related to serotonin (SLC6A4 and TPH2), dopamine (DRD4 and SLC6A3), glutamate (DAOA and DTNBP1), and cell growth and/or maintenance pathways (NRG1, DISC1 and BDNF), although noting a high risk of false positives in the published literature. It was also suggested that individual genes are likely to have only a small effect and to be involved in some aspect related to the disorder (and a broad range of "normal" human behavior) rather than the disorder per se.[31]
Advanced paternal age has been linked to a somewhat increased chance of bipolar disorder in offspring, consistent with a hypothesis of increased new genetic mutations.[32]
Physiological
The causes of bipolar disorder have yet to be fully uncovered, but there is strong evidence that brain structure anomalies play a role.
Abnormalities in the structure and/or function of certain brain circuits could underlie bipolar. Meta-analyses of structural MRI studies in bipolar disorder report an increase in the volume of the lateral ventricles, globus pallidus and increase in the rates of deep white matter hyperintensities.[33][34][35] Functional MRI findings suggest that abnormal modulation between ventral prefrontal and limbic regions, especially the amygdala, likely contribute to poor emotional regulation and mood symptoms.[36]
According to the "kindling" hypothesis, when people who are genetically predisposed toward bipolar disorder experience stressful events, the stress threshold at which mood changes occur becomes progressively lower, until the episodes eventually start (and recur) spontaneously. There is evidence of hypothalamic-pituitary-adrenal axis (HPA axis) abnormalities in bipolar disorder due to stress.[37][38][39][40]
Other brain components which have been proposed to play a role are the mitochondria,[41] and a sodium ATPase pump,[42] causing cyclical periods of poor neuron firing (depression) and hypersensitive neuron firing (mania). This may only apply for type one, but type two apparently results from a large confluence of factors.[citation needed] Circadian rhythms and melatonin activity also seem to be altered.[43]
Environmental
Evidence suggests that environmental factors play a significant role in the development and course of bipolar disorder, and that individual psychosocial variables may interact with genetic dispositions.[31] There is fairly consistent evidence from prospective studies that recent life events and interpersonal relationships contribute to the likelihood of onsets and recurrences of bipolar mood episodes, as they do for onsets and recurrences of unipolar depression.[44] There have been repeated findings that between a third and a half of adults diagnosed with bipolar disorder report traumatic/abusive experiences in childhood, which is associated on average with earlier onset, a worse course, and more co-occurring disorders such as PTSD.[45] The total number of reported stressful events in childhood is higher in those with an adult diagnosis of bipolar spectrum disorder compared to those without, particularly events stemming from a harsh environment rather than from the child's own behavior.[46]
There is nothing that even mentions demonic possession for this or any mental disorder. People usually say that their mental disorder or handicap is caused by demons because of their hatred and bias against the disabled and those with a mental condition and they do not even bother to research it.
Some people claim that this is not written by someone who is saved but on the other hand, they have no right to judge anybody and therefore just because a person is saved does not mean they are incapable of error and being wrong.
It is very sad that there are those who rather judge others than to get to know them.
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AgIn, narrow is the gate and there shall be few who find it, does not automatically mean 'the majority of people are going to Hell'.
Narrow is the gate of righteousness.
In the bible, there were a handful of properly righteous people, but millions who tried to be.
Intent matters. That's why God looks at the heart.
So ask yourself, and be truthful, what is your intent and what purpose does any of this serve? Do not put it on a page because there is no need to prove yourself to me. Only to God. He'll be looking at your heart.
Narrow is the gate of righteousness.
In the bible, there were a handful of properly righteous people, but millions who tried to be.
Intent matters. That's why God looks at the heart.
So ask yourself, and be truthful, what is your intent and what purpose does any of this serve? Do not put it on a page because there is no need to prove yourself to me. Only to God. He'll be looking at your heart.
Shyness
It seems like you are the one hating my friend. You call me Ignorant? Say my thread should be just thrown away because you don’t like it. What happened to freedom of speech? I guess as long as you like it then it can stand as freedom of speech but if you don’t then we must BURN THE BOOK???
I say if you can’t handle the heat then get out the kitchen.
It seems like you are the one hating my friend. You call me Ignorant? Say my thread should be just thrown away because you don’t like it. What happened to freedom of speech? I guess as long as you like it then it can stand as freedom of speech but if you don’t then we must BURN THE BOOK???
I say if you can’t handle the heat then get out the kitchen.
You have two choices in life. You can live by the law where you break one you break them all or you can be saved by grace through faith. With all my sins I chose Grace by faith. I would never make it under the law and none of these so called righteous people you speak of will either
Hell awaits them
Hell awaits them
It does not follow absolutely that "the casting out" (itself) cured them -- Jesus could have simply done that ALSO at the same time... (It is possible.)
"Might have... Might not have..."
I imagine that (because I am not going to look them all up - just going "off of memory" here) - in most, if not all, places in scripture where an account of such is given -- the two things are probably indicated as happening at the same time without saying that the one actually caused the other...
Now, if scripture actually says that the one actually caused the other -- that is a different case...
.
"Might have... Might not have..."
I imagine that (because I am not going to look them all up - just going "off of memory" here) - in most, if not all, places in scripture where an account of such is given -- the two things are probably indicated as happening at the same time without saying that the one actually caused the other...
Now, if scripture actually says that the one actually caused the other -- that is a different case...
.